For research use only. Not for human consumption. Not medical advice — consult a licensed clinician.

Nootropic / Cognitive

P21

Also known as: P021

P21 (also P021) is a small peptidergic compound — the tetrapeptide core Asp-Gly-Gly-Leu with an N-terminal acetyl and a C-terminal adamantylated glycine amide (Ac-DGGL(A)G-NH2) — derived from an active region (residues ~148–151) of human ciliary neurotrophic factor (CNTF). It was designed and characterized in the Khalid Iqbal laboratory at the New York State Institute for Basic Research in Developmental Disabilities (NYS IBR). It is an experimental, preclinical compound — NOT FDA-approved and NOT tested in humans in published trials. Distinct from the cyclin-dependent kinase inhibitor protein p21/CDKN1A/WAF1 (a completely different gene product; the two share only the name).

Where to buy · live market data

Who's worth your money for P21

Every current seller, ranked by independent evidence — Merit Score, latest COA purity, and live $/mg (size-normalized). Prices refresh daily. Rankings are never paid.

Top Merit pick

Highest Merit Score (86) of 2 scored sellers

$11.90/mg

$119 · 10mg · 98.6% purity

Best value

Lowest $/mg among scored sellers

$19.80/mg

$198 · 10mg

no COAs yet

Average price

$15.35/mg

Sellers

10

45-day trend

+9.8%

9 of 10 sellers have a current price· 3 stale hidden· 4 unverified hidden

Prices observed from public storefronts (last 24h), normalized to $/mg. "Evidence" is Merit's 0–100 Merit Score, derived only from observable verification evidence (methodology on /about); "Purity" is the latest independent COA. Some buy links are affiliate links — Merit may earn a commission at no extra cost to you, and where a vendor offers one, the code shown gets you a discount at their checkout. Affiliate status never affects price data, ranking, or the Merit Score (full policy on /disclosure). Research use only.

Watch P21

Get one email when the market actually moves — no newsletter, no noise.

Compound reference

About P21

CAS

1246751-68-7

Molecular formula

C27H42N6O8

Sequence

DGGL

Typical dose range

500–750 mcg/day intranasal (research use); 250–1000 mcg/day range reported

Half-life

>6 hours (rodent; estimated, based on parent Peptide 6 and adamantane-modified stability)

Research depth

20 citations indexed for P21

All research on P21 →

Study · 2026

Single-Cell and Multiomics Characterization of p21 in Cancer Progression and Therapeutic Sensitivity

Introduction CDKN1A (encoding p21) is a canonical regulator of cell cycle arrest and genomic stability. However, its functional spectrum within the tumor microenvironment (TME), especially at single-cell resolution, remains insufficiently defined.

Study · 2026

Exposure to high doses of tyre antioxidant 6PPD causes senescence to induce unexplained miscarriage by suppressing BAZ1B-mediated ubiquitination degradation of P21

Background Unexplained miscarriage (UM) highly occurs and largely limits human reproduction. Cellular senescence is a ubiquitous process that is associated with many human diseases.

Study · 2026

Genomic Disruption of CAMKMT by t(2;11)(p21;q23) Reveals a Glycolytic Reprogramming Mechanism in Acute Myeloid Leukemia

Introduction The t(2;11)(p21;q23) translocation without KMT2A rearrangement has been reported in ~16 hematologic neoplasms, but its molecular target(s) remain unknown. We aimed to identify the genes disrupted by this translocation and to clarify its contribution to Acute Myeloid Leukemia (AML) pathogenesis.

animal · 2026

CRISPR/Cas9 system-mediated p21 knockout impairs the MITF signaling pathway

The CRISPR/Cas9 method facilitates targeted disruption of gene sequences, providing a reliable means to analyze gene-dependent regulatory pathways. This study aims to investigate melanogenesis in p21-knockout B16F1 cells generated by the CRISPR/Cas9 system.

Study · 2026

Ubiquitin-dependent degradation of p27Kip1 and p21Waf1/Cip1 by AMBRA1 ensures G1 and S phase progression and limits replication stress

The cell cycle orchestrates the events that lead to cell replication and division. AMBRA1 interacts with the E3 ubiquitin ligase CRL4DDB1 complex to regulate the stability of D-type cyclins, key regulators of the G1-S phase transition. However, whether AMBRA1 has a role in the S phase remains to be elucidated.

Study · 2026

Transcription-independent induction of rapid-onset senescence is integral to healing

Cellular senescence plays key roles in tissue repair, tumour suppression and ageing. Here we identify a rapid, transcription‑independent senescence response in skin following injury. Within minutes to hours after wounding, skin cells at the edge of injury display hallmark features of senescence.